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| Product Name | Recombinant Human ATP-sensitive inward rectifier potassium channel 10 (KCNJ10), partial |
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| Description | Purity > 90% as determined by SDS-PAGE. MW: 39.8kDa. Cytoplasmic Domain: 165-379aa. N-terminal 6xHis-SUMO-tagged. Homo sapiens (Human). May be responsible for potassium buffering action of glial cells in the brain. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of Extracellular domain potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by Extracellular domain barium and cesium. |
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| Size | 100ug |
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| Concentration | n/a |
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| Applications | RUO |
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| Other Names | ATP-dependent inwardly rectifying potassium channel Kir4.1Inward rectifier K (+) channel Kir1.2; Potassium channel, inwardly rectifying subfamily J member 10 |
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| Gene, Accession, CAS # | KCNJ10, UniProt: P78508 |
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| Catalog # | RPC21555 |
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| Price | |
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| Order / More Info | Recombinant Human ATP-sensitive inward rectifier potassium channel 10 (KCNJ10), partial from BIOMATIK |
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| Product Specific References | n/a |
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